From: Guest

Date: 1/17/08

IN reply to 'a real health threat'.

I can assure you both are a real health threat.
my mother died from the Heidenhain Variant of CJD 'confirmed', and i damn near died from the MRSA, after refusing blood and cadaver bone. go figure. but this case of CJD in question, it could very well have been caused by the USA strain of mad cow disease i.e. BASE, of which the last two case in the USA were. IF anyone would take the time to read the science, the USA mad cow strain i.e. BASE is more like some sub-types of sporadic CJD, NOT the UK nvCJD. Plus, in 2007 alone, 5 cases of the atypical scrapie NOR-98 were documented in 5 different states in the USA, again, this strain of scrapie i.e. the NOR-98 closely resembles that of sporadic CJD, of which is increasing here in the USA. SO, to catigorically state that this case is not related to mad cow, is not accurate. they simply don't know. could be from many TSE sources here in the USA. ...

[Docket No. APHIS-2006-0041] RIN 0579-AC01

[Federal Register: January 9, 2007 (Volume 72, Number 5)]
[Proposed Rules]
[Page 1101-1129]
From the Federal Register Online via GPO Access []

DERIVED FROM BOVINES [Docket No. APHIS-2006-0041] RIN 0579-AC01
Date: January 9, 2007 at 9:08 am PST

Response to Public Comments on the Harvard Risk Assessment of Bovine
Spongiform Encephalopathy Update, October 31, 2005


The United States Department of Agriculture?s Food Safety and Inspection
Service (FSIS) held a public meeting on July 25, 2006 in Washington, D.C. to
present findings from the Harvard Risk Assessment of Bovine Spongiform
Encephalopathy Update, October 31, 2005 (report and model located on the
FSIS website:

Comments on technical aspects of the risk assessment were then submitted to
FSIS. Comments were received from Food and Water Watch, Food Animal Concerns
Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S.
Singeltary. This document provides itemized replies to the public comments
received on the 2005 updated Harvard BSE risk assessment. Please bear the
following points in mind:

snip...see full text 33 pages ;

From: Terry S. Singeltary Sr. []
Sent: Monday, July 24, 2006 1:09 PM
To: FSIS RegulationsComments
Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE)

Page 1 of 98


Greetings FSIS,

I would kindly like to comment on the following ;

full text of this submission is @ ;

There is a growing number of human CJD cases, and they were presented last
week in San Francisco by Luigi Gambatti(?) from his CJD surveillance

He estimates that it may be up to 14 or 15 persons which display selectively
SPRPSC and practically no detected RPRPSC proteins.


MARCH 26, 2003

RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob

disease in the United States

Email Terry S. Singeltary:

I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to

comment on the CDC's attempts to monitor the occurrence of emerging

forms of CJD. Asante, Collinge et al [1] have reported that BSE

transmission to the 129-methionine genotype can lead to an alternate

phenotype that is indistinguishable from type 2 PrPSc, the commonest

sporadic CJD. However, CJD and all human TSEs are not reportable

nationally. CJD and all human TSEs must be made reportable in every

state and internationally. I hope that the CDC does not continue to

expect us to still believe that the 85%+ of all CJD cases which are

sporadic are all spontaneous, without route/source. We have many TSEs in

the USA in both animal and man. CWD in deer/elk is spreading rapidly and

CWD does transmit to mink, ferret, cattle, and squirrel monkey by

intracerebral inoculation. With the known incubation periods in other

TSEs, oral transmission studies of CWD may take much longer. Every

victim/family of CJD/TSEs should be asked about route and source of this

agent. To prolong this will only spread the agent and needlessly expose

others. In light of the findings of Asante and Collinge et al, there

should be drastic measures to safeguard the medical and surgical arena

from sporadic CJDs and all human TSEs. I only ponder how many sporadic

CJDs in the USA are type 2 PrPSc?

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

Singeltary, Sr et al. JAMA.2001; 285: 733-734.

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in
the United States

Creutzfeldt Jakob Disease


SEAC 99th meeting on Friday 14th December 2007

vCJD case study highlights blood transfusion risk

vCJD transfusion-associated Fourth Case UK

risk factors for sporadic CJD

Creutzfeldt-Jakob Disease, Prion Protein Gene Codon 129VV, and a Novel PrPSc
Type in a Young British Woman

BSE (Mad Cow) Update: Do Reports of sCJD Clusters Matter?

snip... see full text ;

Hardcover, 304 pages plus photos and illustrations.
ISBN 0-387-95508-9 June 2003
BY Philip Yam


Answering critics like Terry Singeltary, who feels that the U.S.
under-counts CJD, Schonberger conceded that the current surveillance system
has errors but stated that most of the errors will be confined to the older

doi:10.1016/S1473-3099(03)00715-1Copyright ? 2003 Published by Elsevier Ltd.


Tracking spongiform encephalopathies in North America

Xavier Bosch

Available online 29 July 2003.
Volume 3, Issue 8, August 2003, Page 463

"My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my
mom to hvCJD (Heidenhain variant CJD) and have been searching for answers
ever since. What I have found is that we have not been told the truth. CWD
in deer and elk is a small portion of a much bigger problem." ...


Terry S. Singeltary Sr.